| Neurogenic inflammation is a process by which sensory nerves may secrete inflammatory mediators, resulting in hyperalgesia and inflammation. |
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| Prostaglandins promote the development of hyperalgesia by sensitizing nociceptive neurons. |
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| Intravenous opiates or ketamine administered before incision can lead to decreases in wound hyperalgesia days after the surgery. |
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| These mediators and the related tissue injury prolong pain and hyperalgesia. |
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| The mechanism for the pain may involve the development of an area of hyperalgesia as a result of myofascial stretch injury. |
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| When patients receive narcotics for long periods, they can even become more sensitive to pain, a condition called hyperalgesia. |
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| Inhibition of inflammatory hyperalgesia was also demonstrated in rats. |
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| Combined action of vasoactive amines and bradykinin mediates allergen-evoked thermal hyperalgesia in rats. |
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| Myoclonus, delirium, hallucinations, hyperalgesia and allodynia are features of opioid-induced neurotoxicity. |
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| Pre-emptive analgesia is preferable to reactive analgesia when conducting surgical procedures, reducing or preventing hyperalgesia, allodynia, or wind-up. |
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| In addition, secondary hyperalgesia has many features or mechanisms related to memory at supraspinal sites. |
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| This may be accompanied by hyperalgesia, allodynia, dysaesthesia or paraesthesiae. |
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| Referred pains, areas of secondary hyperalgesia, autonomic signs, and myospastic activity in otherwise normal structures are expected associated symptoms. |
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