Insufficient maternal intake of folic acid is thought to increase the likelihood of anencephaly as well as other neural tube defects. |
|
The disorder is accompanied by severe spinal defects and often brain and face malformations, such as anencephaly and cyclopia. |
|
Another kind of neural tube defect is anencephaly where the brain is almost completely absent. |
|
Failure of closure typically at the cranial end of the neural tube results in anencephaly, or at the caudal neuropore end, in spina bifida. |
|
The most common neural tube defects diagnosed at birth are spina bifida and anencephaly. |
|
The birth prevalence of both anencephaly and spina bifida has decreased over the past decade. |
|
Unfortunately, there is no known treatment for a child with an anencephaly. |
|
Two common types of open neural tube defects are anencephaly and spina bifida. |
|
Infants born with anencephaly die at or soon after birth. |
|
It is still not known what causes anencephaly. |
|
There is little doubt that taking folic acid tablets in early pregnancy helps to prevent severe neural tube defects, such as spina bifida and anencephaly, a lack of brain growth. |
|
The first abortion was four months from pregnancy resulting in a fetus with anencephaly, kyphosis and cephalocele and the second was a blighted ovum at 3rd month of pregnancy. |
|
Anencephaly results in babies born with underdeveloped brains and incomplete skulls, and most of these children die at birth. |
|
Anencephaly and unilateral anophthalmia were observed in one fetus, microophthalmia was observed in another fetus, and cleft palate was observed in a third fetus. |
|